L
Late-Onset Multiple Carboxylase Deficiency
Posted on July 24, 2011 |
The late onset form of MULTIPLE CARBOXYLASE DEFICIENCY (deficiency of the activities of biotin-dependent enzymes propionyl-CoA carboxylase, methylcrotonyl-CoA carboxylase, and PYRUVATE CARBOXYLASE) due to a defect or deficiency in biotinidase which is essential for recycling BIOTIN.
Latency (Psychology) Period
Posted on July 24, 2011 |
The period from about 5 to 7 years to adolescence when there is an apparent cessation of psychosexual development.
Lateral Thalamic Nucleus
Posted on July 24, 2011 |
A narrow strip of cell groups on the dorsomedial surface of the thalamus. It includes the lateral dorsal nucleus, lateral posterior nucleus, and the PULVINAR.
Lateral Sinus Septic Phlebitis
Posted on July 24, 2011 |
Infectious or noninfectious thrombosis of the lateral sinus of the brain. Septic thrombosis may be associated with OTITIS MEDIA or MASTOIDITIS. CRANIOCEREBRAL TRAUMA; BRAIN NEOPLASMS; NEUROSURGICAL PROCEDURES; THROMBOPHILIA; and other conditions may result in non-septic thrombosis. Clinical manifestations include headache, vertigo, and increased intracranial pressure. Extension of the thrombus into adjacent venous structures may result in INTRACRANIAL HEMORRHAGES or SEIZURES. (From Joynt, Clinical Neurology, 1997, Ch23, pp60-8)
L Isomer Rhamnose
Posted on July 24, 2011 |
A methylpentose whose L- isomer is found naturally in many plant glycosides and some gram-negative bacterial lipopolysaccharides.
L Isomer Phenylalanine
Posted on July 24, 2011 |
An essential aromatic amino acid that is a precursor of MELANIN, DOPAMINE, noradrenalin (NOREPINEPHRINE), and THYROXINE.
L Isomer Norleucine
Posted on July 24, 2011 |
An unnatural amino acid that is used experimentally to study protein structure and function. It is structurally similar to METHIONINE, however it does not contain SULFUR.
Lateral Medullary Syndromes
Posted on July 24, 2011 |
Infarction of the dorsolateral aspect of the medulla due to occlusion of the vertebral artery and/or the posterior inferior cerebellar artery. Clinical manifestations vary with the size of infarction, but may include loss of pain and temperature sensation in the ipsilateral face and contralateral body below the chin; ipsilateral HORNER SYNDROME; ipsilateral ATAXIA; DYSARTHRIA; VERTIGO; nausea, hiccup; dysphagia; and VOCAL CORD PARALYSIS. (From Adams et al., Principles of Neurology, 6th ed, p801)
Lead, Childhood Nervous System Poisoning
Posted on July 24, 2011 |
Neurologic disorders occurring in children following lead exposure. The most frequent manifestation of childhood lead toxicity is an encephalopathy associated with chronic ingestion of lead that usually presents between the ages of 1 and 3 years. Clinical manifestations include behavioral changes followed by lethargy; CONVULSIONS; HALLUCINATIONS; DELIRIUM; ATAXIA; and vomiting. Elevated intracranial pressure (HYPERTENSION, INTRACRANIAL) and CEREBRAL EDEMA may occur. (From Adams et al., Principles of Neurology, 6th ed, p1210-2)
Lead, Adult Neurotoxicity Syndrome
Posted on July 24, 2011 |
Neurologic conditions in adults associated with acute or chronic exposure to lead or any of its salts. The most common lead related neurologic syndrome in adults consists of a polyneuropathy involving motor fibers. This tends to affect distal nerves and may present as wrist drop due to RADIAL NEUROPATHY. Additional features of chronic lead exposure include ANEMIA; CONSTIPATION; colicky abdominal pain; a bluish lead line of the gums; interstitial nephritis (NEPHRITIS, INTERSTITIAL); and saturnine gout. An encephalopathy may rarely occur. (From Adams et al., Principles of Neurology, 6th ed, p1212)